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Adult-onset obesity is triggered by impaired mitochondrial gene expression

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Mitochondrial dysfunction resulting from chronic inflammation has been shown to induce lipid accumulation 58 , whereas IL-6 enhances hepatic triglyceride and glucose levels, thereby exerting pathogenicity in age-related disease The tissue-specific differences in mTOR signaling likely reflect their different energy demands, because mTOR has been shown to respond directly to changes in ATP levels 50 such that the liver inhibits mTOR signaling as a means to preserve energy expenditure that is not present in excess, whereas the heart up-regulates mTOR signaling to cope with potential decline in mitochondrial OXPHOS. Perks , 1 Nicola Ferreira , 1 Tara R.

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